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  • Source: Molecular Endocrinology. Unidade: IFSC

    Subjects: BIOTECNOLOGIA, DOENÇAS (TRATAMENTO)

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    • ABNT

      FATTORI, Juliana et al. RXR agonist modulates TR: corepressor dissociation upon 9-cis retinoic acid treatment. Molecular Endocrinology, v. 29, n. 2, p. 258-273, 2015Tradução . . Disponível em: https://doi.org/10.1210/me.2014-125. Acesso em: 01 maio 2024.
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      Fattori, J., Campos, J. L. O., Doratioto, T. R., Assis, L. M., Vitorino, M. T., Polikarpov, I., & Xavier-Neto, J. (2015). RXR agonist modulates TR: corepressor dissociation upon 9-cis retinoic acid treatment. Molecular Endocrinology, 29( 2), 258-273. doi:10.1210/me.2014-125
    • NLM

      Fattori J, Campos JLO, Doratioto TR, Assis LM, Vitorino MT, Polikarpov I, Xavier-Neto J. RXR agonist modulates TR: corepressor dissociation upon 9-cis retinoic acid treatment [Internet]. Molecular Endocrinology. 2015 ; 29( 2): 258-273.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2014-125
    • Vancouver

      Fattori J, Campos JLO, Doratioto TR, Assis LM, Vitorino MT, Polikarpov I, Xavier-Neto J. RXR agonist modulates TR: corepressor dissociation upon 9-cis retinoic acid treatment [Internet]. Molecular Endocrinology. 2015 ; 29( 2): 258-273.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2014-125
  • Source: Molecular Endocrinology. Unidade: IFSC

    Subjects: NANOPARTÍCULAS, HORMÔNIOS TIREOIDIANOS, RECEPTORES HORMONAIS

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      SOUZA, P. C. T. et al. Identification of a new hormone-binding site on the surface of thyroid hormone receptor. Molecular Endocrinology, v. 28, n. 4, p. 534-545, 2014Tradução . . Disponível em: https://doi.org/10.1210/me.2013-1359. Acesso em: 01 maio 2024.
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      Souza, P. C. T., Puhl, A. C., Martínez, L., Aparício, R., Nascimento, A. S., Figueira, A. C. M., et al. (2014). Identification of a new hormone-binding site on the surface of thyroid hormone receptor. Molecular Endocrinology, 28( 4), 534-545. doi:10.1210/me.2013-1359
    • NLM

      Souza PCT, Puhl AC, Martínez L, Aparício R, Nascimento AS, Figueira ACM, Nguyen P, Webb P, Skaf MS, Polikarpov I. Identification of a new hormone-binding site on the surface of thyroid hormone receptor [Internet]. Molecular Endocrinology. 2014 ; 28( 4): 534-545.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2013-1359
    • Vancouver

      Souza PCT, Puhl AC, Martínez L, Aparício R, Nascimento AS, Figueira ACM, Nguyen P, Webb P, Skaf MS, Polikarpov I. Identification of a new hormone-binding site on the surface of thyroid hormone receptor [Internet]. Molecular Endocrinology. 2014 ; 28( 4): 534-545.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2013-1359
  • Source: Molecular Endocrinology. Unidade: ICB

    Assunto: ANATOMIA

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      ROZANSKI, Andrei et al. M-Protein is down-regulated in cardiac hypertrophy driven by thyroid hormone in rats. Molecular Endocrinology, v. 27, n. 12, p. 2055-2065, 2013Tradução . . Disponível em: https://doi.org/10.1210/me.2013-1018. Acesso em: 01 maio 2024.
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      Rozanski, A., Takano, A. P. C., Kato, P. N., Soares, A. G., Lellis-Santos, C., Campos, J. C., et al. (2013). M-Protein is down-regulated in cardiac hypertrophy driven by thyroid hormone in rats. Molecular Endocrinology, 27( 12), 2055-2065. doi:10.1210/me.2013-1018
    • NLM

      Rozanski A, Takano APC, Kato PN, Soares AG, Lellis-Santos C, Campos JC, Ferreira JCB, Barreto-Chaves MLM, Moriscot AS. M-Protein is down-regulated in cardiac hypertrophy driven by thyroid hormone in rats [Internet]. Molecular Endocrinology. 2013 ; 27( 12): 2055-2065.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2013-1018
    • Vancouver

      Rozanski A, Takano APC, Kato PN, Soares AG, Lellis-Santos C, Campos JC, Ferreira JCB, Barreto-Chaves MLM, Moriscot AS. M-Protein is down-regulated in cardiac hypertrophy driven by thyroid hormone in rats [Internet]. Molecular Endocrinology. 2013 ; 27( 12): 2055-2065.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2013-1018
  • Source: Molecular Endocrinology. Unidade: FM

    Subjects: BIOLOGIA MOLECULAR (PROCESSOS), CÉLULAS DENDRÍTICAS (ANÁLISE), HIPOGONADISMO (GENÉTICA), MUTAÇÃO GENÉTICA, VETORES

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    • ABNT

      ABREU, Ana Paula et al. Evidence of the Importance of the First Intracellular Loop of Prokineticin Receptor 2 in Receptor Function. Molecular Endocrinology, v. 26, n. 8, p. 1417-1427, 2012Tradução . . Disponível em: http://intl-mend.endojournals.org/content/26/8/1417.full.pdf+html. Acesso em: 01 maio 2024.
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      Abreu, A. P., Noel, S. D., Xu, S., Carroll, R. S., Latronico, A. C., & Kaiser, U. B. (2012). Evidence of the Importance of the First Intracellular Loop of Prokineticin Receptor 2 in Receptor Function. Molecular Endocrinology, 26( 8), 1417-1427. doi:10.1210/me.2012-1102
    • NLM

      Abreu AP, Noel SD, Xu S, Carroll RS, Latronico AC, Kaiser UB. Evidence of the Importance of the First Intracellular Loop of Prokineticin Receptor 2 in Receptor Function [Internet]. Molecular Endocrinology. 2012 ; 26( 8): 1417-1427.[citado 2024 maio 01 ] Available from: http://intl-mend.endojournals.org/content/26/8/1417.full.pdf+html
    • Vancouver

      Abreu AP, Noel SD, Xu S, Carroll RS, Latronico AC, Kaiser UB. Evidence of the Importance of the First Intracellular Loop of Prokineticin Receptor 2 in Receptor Function [Internet]. Molecular Endocrinology. 2012 ; 26( 8): 1417-1427.[citado 2024 maio 01 ] Available from: http://intl-mend.endojournals.org/content/26/8/1417.full.pdf+html
  • Source: Molecular Endocrinology. Unidade: ICB

    Assunto: HISTOLOGIA

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      LEONI, Suzana G. et al. Regulation of thyroid oxidative state by thioredoxin reductase has a crucial role in thyroid responses to iodide excess. Molecular Endocrinology, v. 25, n. 11, p. 1924-1935, 2011Tradução . . Disponível em: https://doi.org/10.1210/me.2011-0038. Acesso em: 01 maio 2024.
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      Leoni, S. G., Kimura, E. T., Santisteban, P., & De la Vieja, A. (2011). Regulation of thyroid oxidative state by thioredoxin reductase has a crucial role in thyroid responses to iodide excess. Molecular Endocrinology, 25( 11), 1924-1935. doi:10.1210/me.2011-0038
    • NLM

      Leoni SG, Kimura ET, Santisteban P, De la Vieja A. Regulation of thyroid oxidative state by thioredoxin reductase has a crucial role in thyroid responses to iodide excess [Internet]. Molecular Endocrinology. 2011 ; 25( 11): 1924-1935.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2011-0038
    • Vancouver

      Leoni SG, Kimura ET, Santisteban P, De la Vieja A. Regulation of thyroid oxidative state by thioredoxin reductase has a crucial role in thyroid responses to iodide excess [Internet]. Molecular Endocrinology. 2011 ; 25( 11): 1924-1935.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2011-0038
  • Source: Molecular Endocrinology. Unidade: IFSC

    Subjects: HORMÔNIOS TIREOIDIANOS, RECEPTORES HORMONAIS, LIGANTES, MOLÉCULA

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      FIGUEIRA, A. C. M. et al. Analysis of agonist and antagonist effects on thyroid hormone receptor conformation by hydrogen/deuterium exchange. Molecular Endocrinology, v. 25, n. Ja 2011, p. 15-31, 2011Tradução . . Disponível em: https://doi.org/10.1210/me.2010-0202. Acesso em: 01 maio 2024.
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      Figueira, A. C. M., Saidemberg, D. M., Souza, P. C. T., Martínez, L., Scanlan, T. S., Baxter, J. D., et al. (2011). Analysis of agonist and antagonist effects on thyroid hormone receptor conformation by hydrogen/deuterium exchange. Molecular Endocrinology, 25( Ja 2011), 15-31. doi:10.1210/me.2010-0202
    • NLM

      Figueira ACM, Saidemberg DM, Souza PCT, Martínez L, Scanlan TS, Baxter JD, Skaf MS, Palma MS, Webb P, Polikarpov I. Analysis of agonist and antagonist effects on thyroid hormone receptor conformation by hydrogen/deuterium exchange [Internet]. Molecular Endocrinology. 2011 ; 25( Ja 2011): 15-31.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2010-0202
    • Vancouver

      Figueira ACM, Saidemberg DM, Souza PCT, Martínez L, Scanlan TS, Baxter JD, Skaf MS, Palma MS, Webb P, Polikarpov I. Analysis of agonist and antagonist effects on thyroid hormone receptor conformation by hydrogen/deuterium exchange [Internet]. Molecular Endocrinology. 2011 ; 25( Ja 2011): 15-31.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2010-0202
  • Source: Molecular Endocrinology. Unidade: IFSC

    Subjects: RECEPTORES, ENZIMAS (ATIVIDADE), HORMÔNIOS TIREOIDIANOS, ÁCIDOS, ENDOCRINOLOGIA

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      SONODA, Milton Taidi et al. Ligand dissociation from estrogen receptor is mediated by receptor dimerization: evidence from molecular dynamics simulations. Molecular Endocrinology, v. 22, n. 7, p. 1565-1578, 2008Tradução . . Disponível em: https://doi.org/10.1210/me.2007-0501. Acesso em: 01 maio 2024.
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      Sonoda, M. T., Martínez, L., Webb, P., Skaf, M. S., & Polikarpov, I. (2008). Ligand dissociation from estrogen receptor is mediated by receptor dimerization: evidence from molecular dynamics simulations. Molecular Endocrinology, 22( 7), 1565-1578. doi:10.1210/me.2007-0501
    • NLM

      Sonoda MT, Martínez L, Webb P, Skaf MS, Polikarpov I. Ligand dissociation from estrogen receptor is mediated by receptor dimerization: evidence from molecular dynamics simulations [Internet]. Molecular Endocrinology. 2008 ; 22( 7): 1565-1578.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2007-0501
    • Vancouver

      Sonoda MT, Martínez L, Webb P, Skaf MS, Polikarpov I. Ligand dissociation from estrogen receptor is mediated by receptor dimerization: evidence from molecular dynamics simulations [Internet]. Molecular Endocrinology. 2008 ; 22( 7): 1565-1578.[citado 2024 maio 01 ] Available from: https://doi.org/10.1210/me.2007-0501
  • Source: Molecular Endocrinology. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      BOTERO, D. et al. Ubc6p and ubc7p are required for normal and substrate-induced endoplasmic reticulum associated degradation of the human selenoprotein type 2 iodothyronine monodeiodinase. Molecular Endocrinology, v. 16, p. 1999-2007, 2002Tradução . . Acesso em: 01 maio 2024.
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      Botero, D., Gereben, B., Goncalves, C., Jesus, L. A., Harney, J. W., & Bianco, A. C. (2002). Ubc6p and ubc7p are required for normal and substrate-induced endoplasmic reticulum associated degradation of the human selenoprotein type 2 iodothyronine monodeiodinase. Molecular Endocrinology, 16, 1999-2007.
    • NLM

      Botero D, Gereben B, Goncalves C, Jesus LA, Harney JW, Bianco AC. Ubc6p and ubc7p are required for normal and substrate-induced endoplasmic reticulum associated degradation of the human selenoprotein type 2 iodothyronine monodeiodinase. Molecular Endocrinology. 2002 ; 16 1999-2007.[citado 2024 maio 01 ]
    • Vancouver

      Botero D, Gereben B, Goncalves C, Jesus LA, Harney JW, Bianco AC. Ubc6p and ubc7p are required for normal and substrate-induced endoplasmic reticulum associated degradation of the human selenoprotein type 2 iodothyronine monodeiodinase. Molecular Endocrinology. 2002 ; 16 1999-2007.[citado 2024 maio 01 ]
  • Source: Molecular Endocrinology. Unidade: FM

    Assunto: MEDICINA INTERNA

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      MARTENS, John W. M et al. A homozygous mutation in the luteinizing hormone receptor causes partial leydig cell hypoplasia: correlation between receptor activity and phenotype. Molecular Endocrinology, v. 12, n. 6, p. 775-784, 1998Tradução . . Acesso em: 01 maio 2024.
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      Martens, J. W. M., Post, M. V., Abelin, N. M. A., Ezabella, M., Toledo, S. P. A., Brunner, H. G., & Themmen, A. P. N. (1998). A homozygous mutation in the luteinizing hormone receptor causes partial leydig cell hypoplasia: correlation between receptor activity and phenotype. Molecular Endocrinology, 12( 6), 775-784.
    • NLM

      Martens JWM, Post MV, Abelin NMA, Ezabella M, Toledo SPA, Brunner HG, Themmen APN. A homozygous mutation in the luteinizing hormone receptor causes partial leydig cell hypoplasia: correlation between receptor activity and phenotype. Molecular Endocrinology. 1998 ; 12( 6): 775-784.[citado 2024 maio 01 ]
    • Vancouver

      Martens JWM, Post MV, Abelin NMA, Ezabella M, Toledo SPA, Brunner HG, Themmen APN. A homozygous mutation in the luteinizing hormone receptor causes partial leydig cell hypoplasia: correlation between receptor activity and phenotype. Molecular Endocrinology. 1998 ; 12( 6): 775-784.[citado 2024 maio 01 ]
  • Source: Molecular Endocrinology. Unidade: IQ

    Subjects: BIOQUÍMICA, PROTEÍNAS

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      KIMURA, E et al. Induction of fos and jun proteins by adrenocorticoprotein and phorbol ester but not by 3',5'-ciclic adenosine monophosphate derivates. Molecular Endocrinology, v. 7 , p. 1463-71, 1993Tradução . . Acesso em: 01 maio 2024.
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      Kimura, E., Sonobe, M. H., Armelin, M. C. S., & Armelin, H. A. (1993). Induction of fos and jun proteins by adrenocorticoprotein and phorbol ester but not by 3',5'-ciclic adenosine monophosphate derivates. Molecular Endocrinology, 7 , 1463-71.
    • NLM

      Kimura E, Sonobe MH, Armelin MCS, Armelin HA. Induction of fos and jun proteins by adrenocorticoprotein and phorbol ester but not by 3',5'-ciclic adenosine monophosphate derivates. Molecular Endocrinology. 1993 ;7 1463-71.[citado 2024 maio 01 ]
    • Vancouver

      Kimura E, Sonobe MH, Armelin MCS, Armelin HA. Induction of fos and jun proteins by adrenocorticoprotein and phorbol ester but not by 3',5'-ciclic adenosine monophosphate derivates. Molecular Endocrinology. 1993 ;7 1463-71.[citado 2024 maio 01 ]
  • Source: Molecular Endocrinology. Unidade: FMRP

    Assunto: FARMACOLOGIA

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      MURPHY, L J et al. Identification and characterization of a rat decidual insulin like growth factor binding protein cdna. Molecular Endocrinology, v. 4 , n. 2 , p. 329-36, 1990Tradução . . Acesso em: 01 maio 2024.
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      Murphy, L. J., Senevirante, C., Ballejo, G., Croze, F., & Kennedy, T. G. (1990). Identification and characterization of a rat decidual insulin like growth factor binding protein cdna. Molecular Endocrinology, 4 ( 2 ), 329-36.
    • NLM

      Murphy LJ, Senevirante C, Ballejo G, Croze F, Kennedy TG. Identification and characterization of a rat decidual insulin like growth factor binding protein cdna. Molecular Endocrinology. 1990 ;4 ( 2 ): 329-36.[citado 2024 maio 01 ]
    • Vancouver

      Murphy LJ, Senevirante C, Ballejo G, Croze F, Kennedy TG. Identification and characterization of a rat decidual insulin like growth factor binding protein cdna. Molecular Endocrinology. 1990 ;4 ( 2 ): 329-36.[citado 2024 maio 01 ]
  • Source: Molecular Endocrinology. Unidade: ICB

    Subjects: INSULINA (RESISTÊNCIA), GLICOSE

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      GARVEY, W. T. et al. Expression of a glucose transporter gene cloned from brain in cellular models of insulin resistance: dexamethasone decreases transporter mrna in primary cultured adipocytes. Molecular Endocrinology, v. 3 , n. 7 , p. 1132-1141, 1989Tradução . . Acesso em: 01 maio 2024.
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      Garvey, W. T., Huecksteadt, T. P., Lima, F. B., & Birnbaum, M. J. (1989). Expression of a glucose transporter gene cloned from brain in cellular models of insulin resistance: dexamethasone decreases transporter mrna in primary cultured adipocytes. Molecular Endocrinology, 3 ( 7 ), 1132-1141.
    • NLM

      Garvey WT, Huecksteadt TP, Lima FB, Birnbaum MJ. Expression of a glucose transporter gene cloned from brain in cellular models of insulin resistance: dexamethasone decreases transporter mrna in primary cultured adipocytes. Molecular Endocrinology. 1989 ; 3 ( 7 ): 1132-1141.[citado 2024 maio 01 ]
    • Vancouver

      Garvey WT, Huecksteadt TP, Lima FB, Birnbaum MJ. Expression of a glucose transporter gene cloned from brain in cellular models of insulin resistance: dexamethasone decreases transporter mrna in primary cultured adipocytes. Molecular Endocrinology. 1989 ; 3 ( 7 ): 1132-1141.[citado 2024 maio 01 ]
  • Source: Molecular Endocrinology. Unidade: FMRP

    Assunto: BIOQUÍMICA

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      LUBAHN, O B et al. Human androgen receptor: complementary deoxyribonucleic acid cloning sequence analysis and gene expression in prostate. Molecular Endocrinology, v. 2 , n. 12, p. 1265-75, 1988Tradução . . Acesso em: 01 maio 2024.
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      Lubahn, O. B., Joseph, D. R., Sar, M., Tan, J., Higgs, H. N., Larson, R. E., et al. (1988). Human androgen receptor: complementary deoxyribonucleic acid cloning sequence analysis and gene expression in prostate. Molecular Endocrinology, 2 ( 12), 1265-75.
    • NLM

      Lubahn OB, Joseph DR, Sar M, Tan J, Higgs HN, Larson RE, French FS, Wilson EM. Human androgen receptor: complementary deoxyribonucleic acid cloning sequence analysis and gene expression in prostate. Molecular Endocrinology. 1988 ;2 ( 12): 1265-75.[citado 2024 maio 01 ]
    • Vancouver

      Lubahn OB, Joseph DR, Sar M, Tan J, Higgs HN, Larson RE, French FS, Wilson EM. Human androgen receptor: complementary deoxyribonucleic acid cloning sequence analysis and gene expression in prostate. Molecular Endocrinology. 1988 ;2 ( 12): 1265-75.[citado 2024 maio 01 ]

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